Continue Log out. Please enter a valid email address. Understanding how these cells aid HIV could lead to... Scientists therefore have sought to develop anti-HIV drugs that block the virus from binding to CCR5 or otherwise render the receptor inactive. However, these classes of drugs are in the earliest stage of research and are not FDA approved.
Search the archives. In order for viruses to reproduce, they must infect cells in the body.
Binding The outside of HIV has an outer shell envelope of proteins, fats and sugars. The study, which focused on the CCR 5 receptor, was supported by both U. This RNA is then used by other enzymes to build a new protein or enzyme. Stay Logged In? The outside of HIV has an outer shell envelope of proteins, fats and sugars. Viruses are not technically alive: ScienceDaily shares links with scholarly publications in the TrendMD network and earns revenue from third-party advertisers, where indicated.
That opens up more cell types to HIV infection, and the further spread of the virus inside the body is liable to speed up the disease progression towards full-blown AIDS and death.
View all the latest top news in the environmental sciences, or browse the topics below:. Your body constantly makes new skin and blood cells, and each cell often makes new proteins in order to stay alive and reproduce.
The new HIV then takes some time to mature, which can go on to infect new cells. Although the acquired immunodeficiency syndrome AIDS -causing virus was initially discovered to infect cells via another receptor, CD 4 , researchers found in 1996 that HIV infection also requires a co-receptor — usually CCR 5 , which sits alongside CD 4 on a variety of immune cells.
Retrieved February 22, 2019 from www. RNA is like the construction boss. Please watch for email s from us to confirm your subscription to your selected newsletter s. Journal Reference: ScienceDaily, 12 September 2013.
The new data suggest that the distinction between CCR 5 and CXCR 4 as co-receptors for HIV infection boils down to relatively subtle differences in structural shapes and electric charge distributions in the HIV binding region — differences that will be of interest to HIV drug developers. Once infected, a cell can produce hundreds of new copies of HIV.